How To Reggie Bush Jersey Promote Your Online Businesses ...

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HP Photosmart 7520 e-All-in-One

Best understood as a home printer with a surprising number of office-centric features, the HP Photosmart 7520 e-All-in-One ($199.99 direct) can serve nicely as a home printer, home office printer, or both. It lacks an Ethernet port, but supports Wi-Fi for connection to a network, making it reasonably easy to share, and it delivers on both office-centric and photocentric features.

The 7520 is the next step up in HP's current line from the HP Photosmart 6520 e-All-in-One that I recently reviewed. Not surprisingly, it shares a lot of the same features, and adds a few more.

Under shared features, for example, it includes automatic duplexing (for printing on both sides of a page) and a touch-screen control panel, although the 7520's screen is larger, at 4.33 inches. Also very much worth mention in this category is HP Wireless Direct, HP's enhanced version of Wi-Fi Direct, which makes it easy to connect directly to the printer from a smartphone, tablet, or laptop.

Added features in the 7520 include a 25-sheet automatic document feeder, for scanning multi-page documents and legal size pages easily, and a built-in ability to fax, both directly from the front panel and from a computer, including over a network. Note too that the automatic document feeder allows manual duplexing for copying and scanning (but not for faxing), which lets you easily scan duplex originals to a file on your computer as well as copy both simplex and duplex originals to your choice of simplex or duplex copies.

Despite the similarities to the HP 6520, the 7520 is not simply the same printer with a few additions. It has a fundamentally different ink system that affects both print speed and quality. Unlike the 6520, it uses five ink cartridges rather than four, adding a photo black ink to the standard matte black, a trick that's meant to help it print better looking photos.

Basics and e-Basics
In addition to faxing, the 7520 can print, scan, and copy. It can also print from, and scan directly to, memory cards and USB memory keys, and let you preview photos before printing on its 4.33-inch color display. It does not let you print directly from PictBridge cameras however.

Paper capacity is not a strong point. The meager 125 sheets for the input tray limits the printer to light-duty use even by home office standards. However, it's helped a little by a separate 20-sheet photo tray for photo paper up to 5 by 7 inches. At least you won't have to swap out the paper every time you switch between photos and documents.

Being an e-All-in-One means the 7520 also works with HP Web Apps and HP's version of cloud printing as well as other mobile apps, including Apple AirPrint and the HP ePrint Home & Biz print app (for printing from both Android and iOS devices).

Unfortunately, as with too many of HP's e-All-in-One inkjets, the 7520 doesn't offer an Ethernet connection for a wired network. That means it has to connect by Wi-Fi to use ePrint, AirPrint, or HP's Web apps, with ePrint and Web apps also needing an Internet connection to the network. If you prefer to avoid Wi-Fi on your home network for security reasons, that's a problem, since you can't use any of these features over a USB connection. One small consolation is that HP says the HP ePrint Home & Biz print app can work with an HP Wireless Direct connection to the printer, whether you have a Wi-Fi network or not.

Setup, Speed, and Output Quality
For my tests, I connected the 7520 by USB cable to a Windows Vista system. Setup was standard fare. On our business applications suite, (using QualityLogic's hardware and software for timing), I clocked the printer at 3.7 pages per minute (ppm), making it a tad faster than the Photosmart 6520, at 3.4 ppm.

HP Photosmart 7520 e-All-in-One

This actually makes the 7520 faster than some (very slow) color lasers, but not blazingly fast. The less expensive Editors' Choice Brother MFC-J825DW , for example, managed 4.0 ppm on our tests. However, the 7520 was much faster than the Brother printer for 4 by 6 photos, at 1 minute 7 seconds, leaving the Brother J825DW far behind, at 1:59.

The printer does even better on output quality overall, thanks to above par-quality for text and photos. The text quality falls at the high end of the range that includes the vast majority of inkjet MFPs. As with any inkjet, it doesn't offer quite the crisp, professional look I'd insist on for something like a resume, but it's easily good enough for almost any business or home use otherwise.

Graphics quality is a half-step below par for an inkjet MFP, but still suitable for most home use or internal business needs. Depending on how much of a perfectionist you are, you may or may not consider it good enough for PowerPoint handouts or the like for business use. Color photos, on the other hand, are top tier for an inkjet MFP, which makes them better than what you'll get from most drugstore prints. The printer even did a reasonably good job with black and white photos on my tests, which is what inkjet printers most often have problems with.

Based strictly on its print speed, output quality, and convenience features?including the ADF, touch screen, and the ability to print from and scan to memory cards and USB keys?the HP Photosmart 7520 e-All-in-One is certainly worth considering for a home, a home office, or both. It would earn a more enthusiastic recommendation if also included an Ethernet port, but even without one, it's worth considering whether you have Wi-Fi on your network or not. And if you have Wi-Fi, the Web-related features make it that much more attractive.

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Syrian jet bombs near Turkish border, 6 dead

CEYLANPINAR, Turkey (AP) ? A Syrian fighter jet bombed a rebel-held area near the Turkish border on Monday, killing at least six people and wounding a dozen others, an official said. One rocket-propelled grenade landed in Turkey,

An Associated Press journalist saw the plane bomb an area around the Syrian border town of Ras al-Ayn three times. A Turkish official said one bomb hit a suspected Syrian rebel target about 50 or 60 meters (yards) away from the border with Turkey. Last week Syrian rebels overran three security compounds there and wrestled control of the town, located in Syria's predominantly Kurdish, oil-producing northeastern province of al-Hasaka.

Turkish ambulances ferried 18 wounded Syrians across the border Monday to a hospital in the southeastern Turkish town of Ceylanpinar, a local official said, adding that six of the wounded died. He spoke on condition of anonymity because he was not authorized to speak to reporters. He said the death toll from the attack was expected to rise.

Hours later, a Syrian helicopter was seen flying over Ras al-Ayn, prompting rebels to fire on it with machine guns. The helicopter returned fire but it was not clear if there were any casualties.

The violence in Syria has killed more than 36,000 people since an uprising against President Bashar Assad's regime began in March 2011. Hundreds of thousands have fled the fighting into neighboring Turkey, Jordan, Lebanon and Iraq.

A surge of 11,000 more Syrians escaped into Turkey on Friday following the fighting at Ras al-Ayn.

Earlier Monday, a rocket-propelled grenade round landed on an empty field near Ceylanpinar. No one was injured, the official said. Turkey has been responding with fire to shells and mortars fired from Syria that land on its territory, but there was no immediate Turkish retaliation, according to the official.

The force of aerial bombing shattered windows in Ceylanpinar, the official said, and several people were injured from broken glass and shrapnel. The private Dogan news agency said a Turkish border soldier was also hurt.

Two ambulances were seen arriving at the hospital with two wounded Syrians, one bloodied and his bare torso peppered with shrapnel. The other had a foot injury. Both were rushed on stretchers through the doors.

Dogan agency video footage showed Syrians scrambling across the border Monday past a barbed wire fence, as Turkish soldiers in helmets, some of them in foxholes, directed them.

Also Monday, a Syrian helicopter bombed rebel positions in an area further south of Ras al-Ayn and the rebels could be heard responding with machine guns, the Turkish official said.

He said the rebels had besieged a Syrian military unit in the region of Esfar Najar and the helicopter was trying to open up an escape route for the Assad regime forces. It was also seen dropping ammunition and food for the soldiers, the official said.

In Geneva, the International Federation of Red Cross and Red Crescent Societies said it anticipates that 170,000 refugees from Syria's civil war will require help in Turkey over the next six months. Around 115,000 Syrian refugees have already found shelter in 14 camps in Turkey, according to Turkish officials, and thousands more are waiting to cross in.

___

Associated Press writers Suzan Fraser in Ankara, Turkey, and John Heilprin in Geneva contributed.

Source: http://news.yahoo.com/syrian-jet-bombs-near-turkish-border-6-dead-124028473.html

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Cilia guide neuronal migration in developing brain

ScienceDaily (Nov. 12, 2012) ? A new study demonstrates the dynamic role cilia play in guiding the migration of neurons in the embryonic brain. Cilia are tiny hair-like structures on the surfaces of cells, but here they are acting more like radio antennae.

In developing mouse embryos, researchers were able to see cilia extending and retracting as neurons migrate. The cilia appear to be receiving signals needed for neurons to find their places.

Genetic mutations that cause the neurodevelopmental disorder Joubert syndrome interfere with these migratory functions of cilia, the researchers show. The finding suggests that problems with neuron migration may explain some aspects of Joubert syndrome patients' symptoms.

The results were published in the journal Developmental Cell.

"The most surprising thing was how dynamic the cilia are," says Tamara Caspary, PhD, assistant professor of human genetics at Emory University School of Medicine. "As interneurons migrate into the developing cerebral cortex, they move in steps. When they pause, we could see the cilia extending, as if the interneurons are trying to figure out where to go next."

The paper is the result of a collaboration between Caspary's laboratory and that of Eva Anton, PhD, professor of cell and molecular physiology at University of North Carolina School of Medicine. First author Holden Higginbotham, formerly a postdoc in Anton's laboratory, is now a faculty member at Brigham Young University in Idaho.

Readers may be familiar with motile cilia, which can be found on a paramecium or in our trachea or reproductive organs. In contrast, primary (non-motile) cilia can be found on almost every cell in the human body, each cell having just one. Ciliopathies are a class of genetic disorders involving defects in cilia, and include kidney and eye diseases as well as Joubert syndrome.

Joubert syndrome affects the development of the cerebellum and brain stem, leading to lack of muscle control, breathing problems, and sometimes intellectual disability.

Caspary's laboratory has been studying Arl13b, a gene mutated in Joubert syndrome. Mutations in Arl13b lead to cilia that are short and stubby.

"It's a useful tool for studying the role of cilia in development, because it doesn't take a sledgehammer to the entire structure," Caspary says.

Exactly how Arl13b contributes to the function of cilia is unclear -- it appears to be involved in protein transport needed for building cilia because when it is hyperactivated, cilia are longer.

Caspary's and Anton's laboratories teamed up to look at neuronal migration in mouse embryos where Arl13b was deleted, but only in some types of neurons. "Interneurons" form connections between other neurons and do not connect to muscles or sensory organs. They observed that when Arl13b was deleted in interneurons, those cells did not migrate properly through the developing cortex of the brain.

The team probed cilia by using mice that produced a red fluorescent protein within cilia. Slices of embryonic brain were examined with a confocal microscope, under conditions where the cells stay alive and continue moving for several hours.

The scientists could see the interneurons migrating in spurts, with the cilia tending to extend and move "like basketball players' arms" when the cells paused. In the Arl13b deleted mice, the cilia did not extend as much and often could be seen only as red dots. Reintroducing Arl13b could rescue these defects, while a form of Arl13b found in Joubert syndrome patients could not.

How defects in cilia contribute to Joubert syndrome is complex; cilia are needed for Hedgehog signaling, machinery that controls embryonic patterning. Caspary says the neuron migration problem may explain the intellectual disability aspect, while Hedgehog defects may explain impaired development of the cerebellum and brainstem.

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Story Source:

The above story is reprinted from materials provided by Emory University, via EurekAlert!, a service of AAAS.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Holden Higginbotham, Tae-Yeon Eom, Laura?E. Mariani, Amelia Bachleda, Joshua Hirt, Vladimir Gukassyan, Corey L. Cusack, Cary Lai, Tamara Caspary, E.S. Anton. Arl13b in Primary Cilia Regulates the Migration and Placement of Interneurons in the Developing Cerebral Cortex. Developmental Cell, 2012; 23 (5): 925 DOI: 10.1016/j.devcel.2012.09.019

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/~3/eUJ6StoG8jU/121112150343.htm

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Another Win For Quantified Self And Big Data Startups: Weight Loss Platform Retrofit Gains An $8M Series A Round Led By DFJ

retrofit logoThe weight loss and health industries have collided head-on with the tech world, with even the most mainstream depictions of startup life pitching products to help people stay trim and out of the doctor's office. In one of the more interesting developments, Retrofit, a Chicago-based weight loss program designed for busy professionals that incorporates peripheral services like Skype, Fitbit and connected, wireless scales, along with live mentorship from "wellness experts," is today announcing that it has picked up $8 million in funding led by?Draper Fisher Jurvetson to take its product national and to new areas of service. The Series A round brings total funding for Retrofit to?$10.7 million, with other investors including Correlation Ventures, Hyde Park Angels, New World Ventures, and I2A Fund.

Source: http://feedproxy.google.com/~r/Techcrunch/~3/r6VV9tm14SM/

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AAA Michigan: Gas prices up 6 cents from last week

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Source: http://news.yahoo.com/aaa-michigan-gas-prices-6-cents-last-week-155957524--finance.html

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How chronic inflammation can cause cancer

ScienceDaily (Nov. 12, 2012) ? A hormone-like substance produced by the body to promote inflammation can cause an aggressive form of leukemia when present at high levels, according to a new study by researchers at the Ohio State University Comprehensive Cancer Center -- Arthur G. James Cancer Hospital and Richard J. Solove Research Institute (OSUCCC -- James).

The study shows that high levels of interleukin-15 (IL-15) alone can cause large granular lymphocytic (LGL) leukemia, a rare and usually fatal form of cancer, in an animal model. The researchers also developed a treatment for the leukemia that showed no discernible side effects in the animal model.

Published in the journal Cancer Cell, the findings show that IL-15 is also overexpressed in patients with LGL leukemia and that it causes similar cellular changes, suggesting that the treatment should also benefit people with the malignancy.

"We know that inflammation can cause cancer, but we don't know the exact mechanism," says principal investigator Dr. Michael A. Caligiuri, CEO of The James Cancer Hospital and Solove Research Institute, and director of Ohio State's Comprehensive Cancer Center.

"Here, we show one way it can happen, and we used that information to potentially cure the cancer."

Normally, the body releases IL-15 to stimulate the development, survival and proliferation of natural-killer cells, which are immune cells that destroy cancer and virus-infected cells. This research shows that when IL-15 is present in high amounts in the body for prolonged periods, such as during chronic inflammation, it can cause certain immune cells called large granular lymphocytes, or LGLs, to become cancerous.

This malignant transformation begins when IL-15 attaches to receptors on the surface of normal LGLs, an event that boosts levels of a cancer-causing protein called Myc (pronounced "mick") inside the cells. The high Myc levels, in turn, bring changes that cause chromosome instability and additional gene mutations. The high Myc levels also activate a process called DNA methylation, which turns off a variety of genes, including important genes that normally suppress cancer growth.

"We stand the best chance of curing cancer when we understand its causes," says first author Anjali Mishra, a postdoctoral researcher in Caligiuri's laboratory. "Once we understood how this inflammatory hormone causes this leukemia, we used that information to develop a treatment by interfering with the process."

Caligiuri and Mishra were joined in this study by Dr. Guido Marcucci, associate director for Translational Research at the OSUCCC -- James, Dr. Robert Lee, professor of pharmaceutics and pharmaceutical chemistry in Ohio State's College of Pharmacy and a group of collaborators. The investigators conducted the research using cells isolated from patients with LGL leukemia and a mouse model of the disease. Key findings include:

  • Exposing normal, human, large granular lymphocytes to IL-15 caused cell proliferation, chromosomal instability and global DNA hypermethylation;
  • Excessive IL-15 activated the cancer-causing Myc oncogene in large granular lymphocytes, leading to genetic instability, DNA hypermethylation and malignant transformation;
  • Details of how Myc upregulation causes the genetic instability and hypermethylation.

Lee developed a liposomal formulation of the proteosome inhibitor bortezomib that shuts down the cancer-causing pathway, potentially curing the malignancy. Leukemic mice treated with the liposomal bortezomib showed 100 percent survival at 130 days versus 100 percent mortality at 60-80 days for control animals.

"We now plan to develop this drug for clinical use," says Marcucci, who holds the John B. and Jane T. McCoy Chair in Cancer Research in Cancer Research.

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Story Source:

The above story is reprinted from materials provided by Ohio State University Medical Center, via EurekAlert!, a service of AAAS.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Anjali Mishra, Shujun Liu, Gregory?H. Sams, Douglas?P. Curphey, Ramasamy Santhanam, Laura?J. Rush, Deanna Schaefer, Lauren?G. Falkenberg, Laura Sullivan, Laura Jaroncyk, Xiaojuan Yang, Harold Fisk, Lai-Chu Wu, Christopher Hickey, Jason?C. Chandler, Yue-Zhong Wu, Nyla?A. Heerema, Kenneth?K. Chan, Danilo Perrotti, Jianying Zhang, Pierluigi Porcu, Frederick?K. Racke, Ramiro Garzon, Robert?J. Lee, Guido Marcucci, Michael?A. Caligiuri. Aberrant Overexpression of IL-15 Initiates Large Granular Lymphocyte Leukemia through Chromosomal Instability and DNA Hypermethylation. Cancer Cell, 2012; 22 (5): 645 DOI: 10.1016/j.ccr.2012.09.009

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/genes/~3/y5UzY2pOmA0/121112135512.htm

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